File Name: bone loss and patterns of bone destruction .zip
- Bone destruction patterns in periodontal disease
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- Patterns of bone destruction in periodontal diseases
Microbial etiology of periodontal diseases is well established, but the present data strongly suggest an equally important role of host immune response in the pathogenesis of periodontal diseases. The initial response to bacterial infection is a local inflammatory reaction that activates the innate immune system, causing the release of an array of cytokines and other mediators and propagation of inflammation through the gingival tissues 1, 2. Due to initiation of bacterial and host immune interaction, destruction of periodontal connective tissue results which continues until the source of infection, i.
By Ashish Jain, Neeta V. Gender-based heterogeneity in periodontal disease has been witnessed in the recent past with huge mounting evidence. The composite effect of sex-based genetic structure and the sex steroid hormones runs in line with the corresponding gender-related differences in risk for chronic periodontitis. Since estrogens, the predominant sex hormones in women, show immune protective and anti-inflammatory effects in hormonally active premenopausal women, they show better periodontal status compared to age-matched men.
Bone destruction patterns in periodontal disease
We apologize for the inconvenience...
Patterns of bone destruction in periodontal diseases
Stefan A. Alveolar bone loss is a hallmark of periodontitis progression and its prevention is a key clinical challenge in periodontal disease treatment. Bone destruction is mediated by the host immune and inflammatory response to the microbial challenge.
Tobacco smoking is an independent risk factor for periodontal disease which increases periodontal pocketing, attachment loss, as well as bone loss leading to varied severity and bone destruction in the form of horizontal and vertical patterns. The aim of the present study is to determine and measure the types and severity of bone destruction in chronic periodontitis CP patients with tobacco smoking habit using intraoral periapical IOPA radiographs and transgingival probing. A total of 60 male participants with CP were included in the study. Group A comprised 30 heavy cigarette smokers and Group B comprised 30 nonsmokers.
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